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Data updated: Mar 10, 2026

ZYMFENTRA

INFLIXIMAB-DYYB
Immunology Approved 2023-10-20

ZYMFENTRA (infliximab-dyyb) is a tumor necrosis factor (TNF) blocker indicated for the maintenance treatment of adults with moderately to severely active ulcerative colitis or Crohn’s disease. The medication is specifically used in patients who have previously received treatment with an intravenous infliximab product. It serves as a therapeutic option for the ongoing management of these chronic inflammatory bowel conditions following initial intravenous therapy.

Source: FDA Label • CELLTRION

How ZYMFENTRA Works

Infliximab-dyyb functions by binding with high affinity to both the soluble and transmembrane forms of tumor necrosis factor alpha (TNFα). This binding action neutralizes the biological activity of TNFα and prevents it from interacting with its cellular receptors. By inhibiting these interactions, the drug blocks the inflammatory signaling pathways associated with TNFα.

Source: FDA Label
1
Indication
--
Phase 3 Trials
2
Years on Market

Details

Status
Prescription
First Approved
2023-10-20
Routes
INJECTION
Dosage Forms
INJECTABLE

Companies

Active Ingredient: INFLIXIMAB-DYYB

ZYMFENTRA Approval History

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What ZYMFENTRA Treats

2 indications

ZYMFENTRA is approved for 2 conditions since its original approval in 2023. These indications span multiple therapeutic areas including oncology, immunology, and more.

  • Ulcerative Colitis
  • Crohn's Disease
Source: FDA Label

ZYMFENTRA Boxed Warning

SERIOUS INFECTIONS and MALIGNANCY See full prescribing information for complete boxed warning Increased risk of serious infections leading to hospitalization or death, including tuberculosis (TB), bacterial sepsis, invasive fungal infections (such as hitoplasmosis) and infections due to ohter opportunistic pathogens. (5.1) Discontinue ZYMFENTRA if a patient develops a serious infection or sepsis. (5.1) Perform test for latent TB; if positive, start treatment for TB prior to starting ZYMFENTRA. M...

ZYMFENTRA Target & Pathway

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Target

TNF (Tumor Necrosis Factor) Cytokine

A pro-inflammatory cytokine that plays a central role in immune responses and inflammation. Excess TNF contributes to autoimmune diseases like rheumatoid arthritis, psoriasis, and inflammatory bowel disease. Blocking TNF reduces inflammation and prevents tissue damage.

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Active Pipeline

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Key Completed Trials

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Completed studies with published results, ranked by significance

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Trial Timeline

Full development history with FDA approval milestones

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Understanding FDA Approval Types
Count Type What it means
- ORIG Original approval - drug first enters market
- SUPPL - Efficacy New indication (new disease/condition approved)
- SUPPL - Labeling Label text changes (warnings, dosing updates)
- SUPPL - Manufacturing Production changes (new facility)
- SUPPL - Chemistry Formulation changes (new dosage strength)

Green lines in the timeline show ORIG and Efficacy approvals - the clinically meaningful milestones.

ZYMFENTRA FDA Label Details

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Indications & Usage

FDA Label (PDF)

ZYMFENTRA is a tumor necrosis factor (TNF) blocker indicated in adults for maintenance treatment of: moderately to severely active ulcerative colitis following treatment with an infliximab product administered intravenously. moderately to severely active Crohn’s disease following with an infliximab products administered intraneously. ZYMFENTRA is indicated in adults for maintenance treatment of: moderately to severely active ulcerative colitis following treatment with an infliximab product administered intravenously. moderately to severely active Crohn's disease following treatment with an inf...

⚠️ BOXED WARNING

WARNING: SERIOUS INFECTIONS and MALIGNANCY See full prescribing information for complete boxed warning Increased risk of serious infections leading to hospitalization or death, including tuberculosis (TB), bacterial sepsis, invasive fungal infections (such as hitoplasmosis) and infections due to oht...

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Data Sources

Data sourced from official FDA and NIH databases. Click links to verify on original sources.