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Pharma Intelligence, Simplified

Data updated: Mar 10, 2026

VABYSMO

FARICIMAB-SVOA
Ophthalmology Approved 2022-01-28

Vabysmo (faricimab-svoa) is a vascular endothelial growth factor (VEGF) and angiopoietin-2 (Ang-2) inhibitor. It is indicated for the treatment of patients with neovascular (wet) age-related macular degeneration, diabetic macular edema, and macular edema following retinal vein occlusion. This therapeutic agent is used to manage retinal conditions where vascular instability and increased permeability are present.

Source: FDA Label • Roche

How VABYSMO Works

Faricimab is a humanized bispecific antibody that functions by binding to and inhibiting both VEGF-A and Ang-2. By blocking VEGF-A, the drug suppresses the proliferation of endothelial cells, the formation of new blood vessels, and vascular leakage. Inhibition of Ang-2 is thought to promote vascular stability and desensitize blood vessels to the effects of VEGF-A. These dual pathways address the increased levels of Ang-2 found in patients with certain macular and retinal vascular diseases.

Source: FDA Label
3
Indications
--
Phase 3 Trials
1
Priority Reviews
4
Years on Market

Details

Status
Prescription
First Approved
2022-01-28
Routes
INTRAVITREAL
Dosage Forms
INJECTABLE

Companies

Active Ingredient: FARICIMAB-SVOA

VABYSMO Approval History

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What VABYSMO Treats

3 indications

VABYSMO is approved for 3 conditions since its original approval in 2022. These indications span multiple therapeutic areas including oncology, immunology, and more.

  • Neovascular Age-Related Macular Degeneration
  • Diabetic Macular Edema
  • Macular Edema Following Retinal Vein Occlusion
Source: FDA Label

VABYSMO Target & Pathway

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Target

VEGF (Vascular Endothelial Growth Factor) Growth Factor

A signaling protein that stimulates the formation of new blood vessels (angiogenesis). Tumors need blood supply to grow, so they secrete VEGF to create new vessels. Blocking VEGF starves tumors of oxygen and nutrients, inhibiting their growth.

Pathway Context

VEGF binds to VEGFR on blood vessel cells to stimulate new vessel formation

VEGFR (Vascular Endothelial Growth Factor Receptor) receptor

Receptors on blood vessel cells that respond to VEGF signals to form new blood vessels. Cancer cells exploit this pathway to ensure blood supply for tumor growth. Blocking VEGFRs prevents tumor angiogenesis and limits cancer progression.

VABYSMO Competitors

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6 other drugs also target VEGF. Compare mechanisms, indications, and trial activity.

Drug = Competitor name Company = Manufacturer N indic. = FDA-approved indications → Date = Patent/exclusivity expiry

Competitors share the same molecular target (VEGF). Earlier expiry dates signal biosimilar/generic opportunities.

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Active Pipeline

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Ongoing clinical trials by development phase

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Key Completed Trials

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Completed studies with published results, ranked by significance

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Trial Timeline

Full development history with FDA approval milestones

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Understanding FDA Approval Types
Count Type What it means
- ORIG Original approval - drug first enters market
- SUPPL - Efficacy New indication (new disease/condition approved)
- SUPPL - Labeling Label text changes (warnings, dosing updates)
- SUPPL - Manufacturing Production changes (new facility)
- SUPPL - Chemistry Formulation changes (new dosage strength)

Green lines in the timeline show ORIG and Efficacy approvals - the clinically meaningful milestones.

VABYSMO FDA Label Details

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Indications & Usage

FDA Label (PDF)

VABYSMO is a vascular endothelial growth factor (VEGF) and angiopoietin 2 (Ang-2) inhibitor indicated for the treatment of patients with: VABYSMO is a vascular endothelial growth factor (VEGF) and angiopoietin-2 (Ang-2) inhibitor indicated for the treatment of patients with: Neovascular (Wet) Age-Related Macular Degeneration (nAMD) Diabetic Macular Edema (DME) Macular Edema Following Retinal Vein Occlusion (RVO) 1.1 Neovascular (wet) Age-Related Macular Degeneration (nAMD) 1.2 Diabetic Macular Edema (DME) 1.3 Macular Edema Following Retinal Vein Occlusion (RVO)

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Data Sources

Data sourced from official FDA and NIH databases. Click links to verify on original sources.